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goat anti human il 17ra ab  (R&D Systems)


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    R&D Systems goat anti human il 17ra ab
    FIGURE 4. Intranasal IL-25 administration did not induce histological signs of lung inflammation in IL-17RB KO or <t>IL-17RA</t> KO mice. WT mice (A and B), IL-17RB KO mice (C and D), and IL-17RA KO mice (E and F) (n 5 per group) were i.n. dosed with vehicle for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. WT mice (G and H), IL-17RB KO mice (I and J), and IL-17RA KO mice (K and L) (n 5 per group) were i.n. dosed with 0.5 g of IL-25 for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. Formalin-fixed lung tissue sections were stained with H&E (A–L) or PAS (M and N) for analysis. Goblet cell hyperplasia was assessed in PAS-stained sections as described in Materials and Methods. Original magnification, 20 in A, C, E, G, I, and K. Original magnification, 400 in B, D, F, H, J, and L. Statistical analyses of WT and KO mice were performed using a nonparametric one-way ANOVA.
    Goat Anti Human Il 17ra Ab, supplied by R&D Systems, used in various techniques. Bioz Stars score: 92/100, based on 10 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/goat anti human il 17ra ab/product/R&D Systems
    Average 92 stars, based on 10 article reviews
    goat anti human il 17ra ab - by Bioz Stars, 2026-03
    92/100 stars

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    1) Product Images from "Identification of functional roles for both IL-17RB and IL-17RA in mediating IL-25-induced activities."

    Article Title: Identification of functional roles for both IL-17RB and IL-17RA in mediating IL-25-induced activities.

    Journal: Journal of immunology (Baltimore, Md. : 1950)

    doi: 10.4049/jimmunol.181.6.4299

    FIGURE 4. Intranasal IL-25 administration did not induce histological signs of lung inflammation in IL-17RB KO or IL-17RA KO mice. WT mice (A and B), IL-17RB KO mice (C and D), and IL-17RA KO mice (E and F) (n 5 per group) were i.n. dosed with vehicle for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. WT mice (G and H), IL-17RB KO mice (I and J), and IL-17RA KO mice (K and L) (n 5 per group) were i.n. dosed with 0.5 g of IL-25 for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. Formalin-fixed lung tissue sections were stained with H&E (A–L) or PAS (M and N) for analysis. Goblet cell hyperplasia was assessed in PAS-stained sections as described in Materials and Methods. Original magnification, 20 in A, C, E, G, I, and K. Original magnification, 400 in B, D, F, H, J, and L. Statistical analyses of WT and KO mice were performed using a nonparametric one-way ANOVA.
    Figure Legend Snippet: FIGURE 4. Intranasal IL-25 administration did not induce histological signs of lung inflammation in IL-17RB KO or IL-17RA KO mice. WT mice (A and B), IL-17RB KO mice (C and D), and IL-17RA KO mice (E and F) (n 5 per group) were i.n. dosed with vehicle for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. WT mice (G and H), IL-17RB KO mice (I and J), and IL-17RA KO mice (K and L) (n 5 per group) were i.n. dosed with 0.5 g of IL-25 for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. Formalin-fixed lung tissue sections were stained with H&E (A–L) or PAS (M and N) for analysis. Goblet cell hyperplasia was assessed in PAS-stained sections as described in Materials and Methods. Original magnification, 20 in A, C, E, G, I, and K. Original magnification, 400 in B, D, F, H, J, and L. Statistical analyses of WT and KO mice were performed using a nonparametric one-way ANOVA.

    Techniques Used: Staining

    FIGURE 6. Abs to IL-17RB, IL-17RA, or IL-25 block IL-25-induced histological signs of pulmonary inflammation. BALB/c mice were i.n. dosed with vehicle (A) or 0.5 g of IL-25 (B–F) for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. Mice (n 5 per group) were treated with control Ab (B) or blocking Abs to IL-17RB (C), IL-25 (D), IL-17RA (E), or IL-17A (F) 3 h before each i.n. dose. Formalin- fixed lung tissue sections were stained with H&E (A–F) or PAS (G) for analysis. A–F, Original magnification, 200. G, Goblet cell hyperplasia was assessed in PAS-stained lung sections as described in Materials and Methods. Statistical analyses were performed using a nonparametric one- way ANOVA.
    Figure Legend Snippet: FIGURE 6. Abs to IL-17RB, IL-17RA, or IL-25 block IL-25-induced histological signs of pulmonary inflammation. BALB/c mice were i.n. dosed with vehicle (A) or 0.5 g of IL-25 (B–F) for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. Mice (n 5 per group) were treated with control Ab (B) or blocking Abs to IL-17RB (C), IL-25 (D), IL-17RA (E), or IL-17A (F) 3 h before each i.n. dose. Formalin- fixed lung tissue sections were stained with H&E (A–F) or PAS (G) for analysis. A–F, Original magnification, 200. G, Goblet cell hyperplasia was assessed in PAS-stained lung sections as described in Materials and Methods. Statistical analyses were performed using a nonparametric one- way ANOVA.

    Techniques Used: Blocking Assay, Control, Staining



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    FIGURE 4. Intranasal IL-25 administration did not induce histological signs of lung inflammation in IL-17RB KO or <t>IL-17RA</t> KO mice. WT mice (A and B), IL-17RB KO mice (C and D), and IL-17RA KO mice (E and F) (n 5 per group) were i.n. dosed with vehicle for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. WT mice (G and H), IL-17RB KO mice (I and J), and IL-17RA KO mice (K and L) (n 5 per group) were i.n. dosed with 0.5 g of IL-25 for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. Formalin-fixed lung tissue sections were stained with H&E (A–L) or PAS (M and N) for analysis. Goblet cell hyperplasia was assessed in PAS-stained sections as described in Materials and Methods. Original magnification, 20 in A, C, E, G, I, and K. Original magnification, 400 in B, D, F, H, J, and L. Statistical analyses of WT and KO mice were performed using a nonparametric one-way ANOVA.
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    FIGURE 4. Intranasal IL-25 administration did not induce histological signs of lung inflammation in IL-17RB KO or <t>IL-17RA</t> KO mice. WT mice (A and B), IL-17RB KO mice (C and D), and IL-17RA KO mice (E and F) (n 5 per group) were i.n. dosed with vehicle for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. WT mice (G and H), IL-17RB KO mice (I and J), and IL-17RA KO mice (K and L) (n 5 per group) were i.n. dosed with 0.5 g of IL-25 for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. Formalin-fixed lung tissue sections were stained with H&E (A–L) or PAS (M and N) for analysis. Goblet cell hyperplasia was assessed in PAS-stained sections as described in Materials and Methods. Original magnification, 20 in A, C, E, G, I, and K. Original magnification, 400 in B, D, F, H, J, and L. Statistical analyses of WT and KO mice were performed using a nonparametric one-way ANOVA.
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    goat anti il 17ra ab  (R&D Systems)
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    FIGURE 4. Intranasal IL-25 administration did not induce histological signs of lung inflammation in IL-17RB KO or <t>IL-17RA</t> KO mice. WT mice (A and B), IL-17RB KO mice (C and D), and IL-17RA KO mice (E and F) (n 5 per group) were i.n. dosed with vehicle for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. WT mice (G and H), IL-17RB KO mice (I and J), and IL-17RA KO mice (K and L) (n 5 per group) were i.n. dosed with 0.5 g of IL-25 for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. Formalin-fixed lung tissue sections were stained with H&E (A–L) or PAS (M and N) for analysis. Goblet cell hyperplasia was assessed in PAS-stained sections as described in Materials and Methods. Original magnification, 20 in A, C, E, G, I, and K. Original magnification, 400 in B, D, F, H, J, and L. Statistical analyses of WT and KO mice were performed using a nonparametric one-way ANOVA.
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    Image Search Results


    FIGURE 4. Intranasal IL-25 administration did not induce histological signs of lung inflammation in IL-17RB KO or IL-17RA KO mice. WT mice (A and B), IL-17RB KO mice (C and D), and IL-17RA KO mice (E and F) (n 5 per group) were i.n. dosed with vehicle for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. WT mice (G and H), IL-17RB KO mice (I and J), and IL-17RA KO mice (K and L) (n 5 per group) were i.n. dosed with 0.5 g of IL-25 for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. Formalin-fixed lung tissue sections were stained with H&E (A–L) or PAS (M and N) for analysis. Goblet cell hyperplasia was assessed in PAS-stained sections as described in Materials and Methods. Original magnification, 20 in A, C, E, G, I, and K. Original magnification, 400 in B, D, F, H, J, and L. Statistical analyses of WT and KO mice were performed using a nonparametric one-way ANOVA.

    Journal: Journal of immunology (Baltimore, Md. : 1950)

    Article Title: Identification of functional roles for both IL-17RB and IL-17RA in mediating IL-25-induced activities.

    doi: 10.4049/jimmunol.181.6.4299

    Figure Lengend Snippet: FIGURE 4. Intranasal IL-25 administration did not induce histological signs of lung inflammation in IL-17RB KO or IL-17RA KO mice. WT mice (A and B), IL-17RB KO mice (C and D), and IL-17RA KO mice (E and F) (n 5 per group) were i.n. dosed with vehicle for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. WT mice (G and H), IL-17RB KO mice (I and J), and IL-17RA KO mice (K and L) (n 5 per group) were i.n. dosed with 0.5 g of IL-25 for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. Formalin-fixed lung tissue sections were stained with H&E (A–L) or PAS (M and N) for analysis. Goblet cell hyperplasia was assessed in PAS-stained sections as described in Materials and Methods. Original magnification, 20 in A, C, E, G, I, and K. Original magnification, 400 in B, D, F, H, J, and L. Statistical analyses of WT and KO mice were performed using a nonparametric one-way ANOVA.

    Article Snippet: The goat anti-human IL-17RA Ab (R&D Systems; catalog no. AF177) and mouse anti-human IL-17A mAb clone 41809 (R&D Systems; catalog no. MAB317) are reported by the manufacturer to inhibit IL-17A-induced IL-6 production by normal human dermal fibroblasts.

    Techniques: Staining

    FIGURE 6. Abs to IL-17RB, IL-17RA, or IL-25 block IL-25-induced histological signs of pulmonary inflammation. BALB/c mice were i.n. dosed with vehicle (A) or 0.5 g of IL-25 (B–F) for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. Mice (n 5 per group) were treated with control Ab (B) or blocking Abs to IL-17RB (C), IL-25 (D), IL-17RA (E), or IL-17A (F) 3 h before each i.n. dose. Formalin- fixed lung tissue sections were stained with H&E (A–F) or PAS (G) for analysis. A–F, Original magnification, 200. G, Goblet cell hyperplasia was assessed in PAS-stained lung sections as described in Materials and Methods. Statistical analyses were performed using a nonparametric one- way ANOVA.

    Journal: Journal of immunology (Baltimore, Md. : 1950)

    Article Title: Identification of functional roles for both IL-17RB and IL-17RA in mediating IL-25-induced activities.

    doi: 10.4049/jimmunol.181.6.4299

    Figure Lengend Snippet: FIGURE 6. Abs to IL-17RB, IL-17RA, or IL-25 block IL-25-induced histological signs of pulmonary inflammation. BALB/c mice were i.n. dosed with vehicle (A) or 0.5 g of IL-25 (B–F) for 4 days, and lungs were harvested for histological analysis 24 h after the final dose. Mice (n 5 per group) were treated with control Ab (B) or blocking Abs to IL-17RB (C), IL-25 (D), IL-17RA (E), or IL-17A (F) 3 h before each i.n. dose. Formalin- fixed lung tissue sections were stained with H&E (A–F) or PAS (G) for analysis. A–F, Original magnification, 200. G, Goblet cell hyperplasia was assessed in PAS-stained lung sections as described in Materials and Methods. Statistical analyses were performed using a nonparametric one- way ANOVA.

    Article Snippet: The goat anti-human IL-17RA Ab (R&D Systems; catalog no. AF177) and mouse anti-human IL-17A mAb clone 41809 (R&D Systems; catalog no. MAB317) are reported by the manufacturer to inhibit IL-17A-induced IL-6 production by normal human dermal fibroblasts.

    Techniques: Blocking Assay, Control, Staining